Hey Sebastian, thanks for your question. Remember, in heart failure, the idea is that you have a decrease in your cardiac output (either because of a decrease in contractility, AKA heart failure with reduced ejection fraction; and/or because of a decrease in preload, AKA heart failure with preserved ejection fraction), which leads to a decrease in your blood pressure. Your body misinterprets the signal, via the baroreceptors, as you having not enough blood volume, and as such, will increase both the sympathetic tone, as well as the renin angiotensin aldosterone system. This is why there’s an increase in ACE, as well as why inhibitors of RAAS as well as beta-blockers are still the major things that actually improve mortality in patients with heart failure.